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Genetic variants in toll-like receptors are not associated with rheumatoid arthritis susceptibility or anti-tumour necrosis factor treatment outcome

Publikation: Bidrag til tidsskriftTidsskriftartikelForskningfagfællebedømt

  • Marieke J H Coenen
  • Christian Enevold
  • Pilar Barrera
  • Mascha M V A P Schijvenaars
  • Erik J M Toonen
  • Hans Scheffer
  • Leonid Padyukov
  • Alf Kastbom
  • Lars Klareskog
  • Anne Barton
  • Wietske Kievit
  • Maarten J Rood
  • Tim L Jansen
  • Dorine Swinkels
  • Piet L C M van Riel
  • Barbara Franke
  • Klaus Bendtzen
  • Timothy R D J Radstake
Several studies point to a role of Toll-like receptors (TLRs) in the development of rheumatoid arthritis (RA). We investigated if genetic variants in TLR genes are associated with RA and response to tumour necrosis factor blocking (anti-TNF) medication.
OriginalsprogEngelsk
TidsskriftP L o S One
Vol/bind5
Udgave nummer12
Sider (fra-til)e14326
ISSN1932-6203
DOI
StatusUdgivet - 1 jan. 2010

ID: 34102844