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Sleep disturbances in IDDM patients with nocturnal hypoglycemia.

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Standard

Sleep disturbances in IDDM patients with nocturnal hypoglycemia. / Bendtson, I; Gade, J; Thomsen, C E; Rosenfalck, A; Wildschiødtz, G.

I: Sleep, Bind 15, Nr. 1, 1992, s. 74-81.

Publikation: Bidrag til tidsskriftTidsskriftartikelForskningfagfællebedømt

Harvard

Bendtson, I, Gade, J, Thomsen, CE, Rosenfalck, A & Wildschiødtz, G 1992, 'Sleep disturbances in IDDM patients with nocturnal hypoglycemia.', Sleep, bind 15, nr. 1, s. 74-81.

APA

Bendtson, I., Gade, J., Thomsen, C. E., Rosenfalck, A., & Wildschiødtz, G. (1992). Sleep disturbances in IDDM patients with nocturnal hypoglycemia. Sleep, 15(1), 74-81.

Vancouver

Bendtson I, Gade J, Thomsen CE, Rosenfalck A, Wildschiødtz G. Sleep disturbances in IDDM patients with nocturnal hypoglycemia. Sleep. 1992;15(1):74-81.

Author

Bendtson, I ; Gade, J ; Thomsen, C E ; Rosenfalck, A ; Wildschiødtz, G. / Sleep disturbances in IDDM patients with nocturnal hypoglycemia. I: Sleep. 1992 ; Bind 15, Nr. 1. s. 74-81.

Bibtex

@article{f61828307daf11dd81b0000ea68e967b,
title = "Sleep disturbances in IDDM patients with nocturnal hypoglycemia.",
abstract = "Eight insulin-dependent diabetic patients were studied to evaluate sleep patterns during normoglycemia and spontaneous and insulin-induced hypoglycemia. Two channels of electroencephalogram (EEG), electromyogram and actooculogram were recorded. The signals were analyzed off-line, using a polygraphic sleep analysis system. The scoring was mainly based on the color density spectral array of the EEG. Blood glucose and growth hormone were measured serially. Asymptomatic, spontaneous nocturnal hypoglycemia occurred in 38{\%} of the nights. Conventional sleep analysis showed a tendency toward prolongation of the two first rapid eye movement cycles on hypoglycemic nights, although it was insufficient to explain the activities seen during hypoglycemia. Blood glucose values below 2.0 mmol/l were observed in some of the patients accompanied by EEG changes with increased theta and delta activity.",
author = "I Bendtson and J Gade and Thomsen, {C E} and A Rosenfalck and G Wildschi{\o}dtz",
note = "Keywords: Adult; Blood Glucose; Cerebral Cortex; Diabetes Mellitus, Type 1; Electroencephalography; Growth Hormone; Humans; Hypoglycemia; Male; Middle Aged; Monitoring, Physiologic; Reaction Time; Signal Processing, Computer-Assisted; Sleep Stages; Sleep, REM",
year = "1992",
language = "English",
volume = "15",
pages = "74--81",
journal = "Sleep (Online)",
issn = "0161-8105",
publisher = "Oxford University Press",
number = "1",

}

RIS

TY - JOUR

T1 - Sleep disturbances in IDDM patients with nocturnal hypoglycemia.

AU - Bendtson, I

AU - Gade, J

AU - Thomsen, C E

AU - Rosenfalck, A

AU - Wildschiødtz, G

N1 - Keywords: Adult; Blood Glucose; Cerebral Cortex; Diabetes Mellitus, Type 1; Electroencephalography; Growth Hormone; Humans; Hypoglycemia; Male; Middle Aged; Monitoring, Physiologic; Reaction Time; Signal Processing, Computer-Assisted; Sleep Stages; Sleep, REM

PY - 1992

Y1 - 1992

N2 - Eight insulin-dependent diabetic patients were studied to evaluate sleep patterns during normoglycemia and spontaneous and insulin-induced hypoglycemia. Two channels of electroencephalogram (EEG), electromyogram and actooculogram were recorded. The signals were analyzed off-line, using a polygraphic sleep analysis system. The scoring was mainly based on the color density spectral array of the EEG. Blood glucose and growth hormone were measured serially. Asymptomatic, spontaneous nocturnal hypoglycemia occurred in 38% of the nights. Conventional sleep analysis showed a tendency toward prolongation of the two first rapid eye movement cycles on hypoglycemic nights, although it was insufficient to explain the activities seen during hypoglycemia. Blood glucose values below 2.0 mmol/l were observed in some of the patients accompanied by EEG changes with increased theta and delta activity.

AB - Eight insulin-dependent diabetic patients were studied to evaluate sleep patterns during normoglycemia and spontaneous and insulin-induced hypoglycemia. Two channels of electroencephalogram (EEG), electromyogram and actooculogram were recorded. The signals were analyzed off-line, using a polygraphic sleep analysis system. The scoring was mainly based on the color density spectral array of the EEG. Blood glucose and growth hormone were measured serially. Asymptomatic, spontaneous nocturnal hypoglycemia occurred in 38% of the nights. Conventional sleep analysis showed a tendency toward prolongation of the two first rapid eye movement cycles on hypoglycemic nights, although it was insufficient to explain the activities seen during hypoglycemia. Blood glucose values below 2.0 mmol/l were observed in some of the patients accompanied by EEG changes with increased theta and delta activity.

M3 - Journal article

C2 - 1557597

VL - 15

SP - 74

EP - 81

JO - Sleep (Online)

JF - Sleep (Online)

SN - 0161-8105

IS - 1

ER -

ID: 5941950